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Factors in the Production and Growth of tumor Metastases
Published on Wednesday, 19 March 2014
Factors in the Production and Growth of tumor Metastases
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- Treatment of R-3327 prostate tumors with a somatostatin analogue (somatuline) as adjuvant therapy following surgical castration
- Simultaneous modulation of COX-2, p300, Akt, and Apaf-1 signaling by melatonin to inhibit proliferation and induce apoptosis in breast cancer cells
- THERAPY WITH SOMATOSTATIN ANALOGUES IN PATIENTS WITH CASTRATION-REFRACTORY PROSTATE CANCER
- Potential role of melatonin in prevention and treatment of oral carcinoma
- Phenotypic changes caused by melatonin increased sensitivity of prostate cancer cells to cytokine-induced apoptosis
- Molecular mechanisms of the pro-apoptotic actions of melatonin in cancer: a review
- Molecular aspects of melatonin (MLT)-mediated therapeutic effects
- Melatonin-Induced Changes in Cytosolic Calcium Might be Responsible for Apoptosis Induction in Tumour Cells
- Melatonin provokes cell death in human B-lymphoma cells by mitochondrial-dependent apoptotic pathway activation
- Melatonin induces pro-apoptotic signaling pathway in human pancreatic carcinoma cells (PANC-1)
- Melatonin enhances hydrogen peroxide-induced apoptosis in human promyelocytic leukaemia HL-60 cells
- Melatonin treatment increases the transcription of cell proliferation-related genes prior to inducing cell death in C6 glioma cells in vitro
- Melatonin suppresses migration and invasion via inhibition of oxidative stress pathway in glioma cells
- Melatonin reduces endothelin-1 expression and secretion in colon cancer cells through the inactivation of FoxO-1 and NF-κβ
- Immunomodulatory effect of melatonin in SK-LU-1 human lung adenocarcinoma cells co-cultured with peripheral blood mononuclear cells
- Vitamin D deficiency is associated with a worse prognosis in metastatic melanoma
- Somatostatin in oncology, the overlooked evidences
- Complete objective response to biological therapy of plurifocal breast carcinoma
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