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Factors in the Production and Growth of tumor Metastases
Published on Wednesday, 19 March 2014
Factors in the Production and Growth of tumor Metastases
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Radioprotective and Radiomitigative Effects of Melatonin in Tissues with Different Proliferative Activity
Somatostatin analog lanreotide in the treatment of castration-resistant prostate cancer (CRPC)
Coexpression of the melatonin receptor 1 and nestin in human breast cancer specimens
Inhibition of breast cancer cell invasion by melatonin is mediated through regulation of the p38 mitogen-activated protein kinase signaling pathway
The effects of growth hormone on therapy resistance in cancer
Anti-apoptotic and antioxidant effects of melatonin protect spleen of whole body γ-irradiated male Sprague-dawley rats
Melatonin decreases breast cancer metastasis by modulating Rho-associated kinase protein-1 expression
Radiomitigation by Melatonin in C57BL/6 Mice: Possible Implications as Adjuvant in Radiotherapy and Chemotherapy
Melatonin for the prevention and treatment of cancer
Melatonin interrupts osteoclast functioning and suppresses tumor-secreted RANKL expression: implications for bone metastases
Mechanisms involved in the pro-apoptotic effect of melatonin in cancer cells
Impact of vitamin D on pathological complete response and survival following neoadjuvant chemotherapy for breast cancer: a retrospective study
Vitamin D sufficiency enhances differentiation of patient-derived prostate epithelial organoids
Apoptosis is triggered by melatonin in an in vivo model of ovarian carcinoma
Melatonin at the Crossroads of Oxidative Stress, Immunity, and Cancer Therapy
Melatonin and breast cancer: cellular mechanisms, clinical studies and future perspectives
Membrane-bound melatonin receptor MT1 down-regulates estrogen responsive genes in breast cancer cells
Melatonin Induces Apoptosis and Inhibits the Proliferation of Cancer Cells via Reactive Oxygen Species-mediated MAPK and mTOR Pathways
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The Di Bella Method (DBM)
About melatonin - 1996
All-trans-retinoic acid and cancer
Biography
The Di Bella Method Increases by the 30% the survival rate for Pancreas tumors and for this reason should be proposed as first line therapy for this type of cancer
Chronic Lymphocytic Leukemia: 4 patients, long-lasting remission
Somatostatin in oncology, the overlooked evidences
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Complete objective response to biological therapy of plurifocal breast carcinoma
Large B-cells Non-Hodgkin's Lymphoma, Stage IV-AE: a Case Report
Non-Hodgkin's Lymphoma, Stage III-B-E: a Case Report
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Declaration recommends eliminate the use of Impact factor for research evaluation
Non–Small-Cell Lung Cancer Patients with Low Performance Status: study on 28 patients
Breast cancer: study on 122 cases. The Di Bella Method (DBM) improved survival, objective response and performance status in a retrospective observational clinical study on 122 cases of breast cancer