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Systemic retinoid therapy for chemoprevention of nonmelanoma skin cancer in a patient treated with vemurafenib
Published on Thursday, 30 October 2014
Systemic retinoid therapy for chemoprevention of nonmelanoma skin cancer in a patient treated with vemurafenib
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- Prolactin inhibitors in oncology
- Activation of the mevalonate pathway in response to anti-cancer treatments drives glioblastoma recurrences through activation of Rac-1
- A Preliminary Phase-2 Study with very High-Dose of Melatonin (1000 mg/day) in Untreatable Advanced Cancer Patients Already Progression on Previous Palliative Therapy with High-Dose Melatonin
- Adjuvant chemotherapy with melatonin for targeting human cancers: A review
- Antitumour activity of melatonin in a mouse model of human prostate cancer: relationship with hypoxia signalling
- Does the use of melatonin overcome drug resistance in cancer chemotherapy?
- Melatonin as a Promising Agent to Treat Ovarian Cancer: Molecular Mechanisms
- Melatonin as an Adjuvant to Antiangiogenic Cancer Treatments
- Long term increased expression of the short form 1b prolactin receptor in PC-3 human prostate cancer cells decreases cell growth and migration, and causes multiple changes in gene expression consistent with reduced invasive capacity
- Short Form 1b Human Prolactin Receptor Down-Regulates Expression of the Long Form
- Co-Administration of Curcumin and Bromocriptine Nano-liposomes for Induction of Apoptosis in Lung Cancer Cells
- Melatonin: an endogenous miraculous indolamine, fights against cancer progression
- Melatonin and breast cancer: Evidences from preclinical and human studies
- Melatonin induces autophagy in neuroblastoma by alleviating Pak2‑mediated endoplasmic reticulum stress
- In vivo dynamic monitoring of self-assembled melatonin nanodrug combined with photothermal effects to alleviate hypoxia to attenuate tumor aggressiveness
- Melatonin: An Endogenous Antiestrogen with Oncostatic Properties
- Modulating effects of famotidine and melatonin on high dose radiation induced cell lethality in normal human and cancer cell lines
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